자료유형 | E-Book |
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개인저자 | Gall, Alevtina A. |
단체저자명 | University of Washington. Molecular and Cellular Biology. |
서명/저자사항 | Defining Mechanisms of Disease Initiation through Analysis of Helicobacter pylori Interactions with Gastric Microbial Communities and the Host. |
발행사항 | [S.l.] : University of Washington., 2018 |
발행사항 | Ann Arbor : ProQuest Dissertations & Theses, 2018 |
형태사항 | 155 p. |
소장본 주기 | School code: 0250. |
ISBN | 9780438175310 |
일반주기 |
Source: Dissertation Abstracts International, Volume: 79-12(E), Section: B.
Adviser: Nina R. Salama. |
요약 | Helicobacter pylori is an exceptionally prevalent bacterial pathogen that colonizes the human stomach where it can cause inflammation, and in some cases lead to gastric ulcers and cancer. H. pylori infection also seems to protect against develo |
요약 | H. pylori may be modulating esophageal adenocarcinoma risk through its interactions with members of the upper gastrointestinal microbial communities. Before that hypothesis can be tested, a comprehensive survey of which microbial community membe |
요약 | H. pylori is recognized by both the innate and adaptive immune system, but this rarely results in bacterial clearance. H. pylori establishes chronic colonization, in part, by evading numerous host defense strategies. However, H. pylori also seem |
요약 | The discrepancy between our in vitro and in vivo findings motivated us to develop a better gastric epithelial cell model to study innate immune signaling pathways involved in H. pylori detection. Using mouse and human gastric tissues, we select |
일반주제명 | Microbiology. Immunology. Cellular biology. |
언어 | 영어 |
기본자료 저록 | Dissertation Abstracts International79-12B(E). Dissertation Abstract International |
대출바로가기 | http://www.riss.kr/pdu/ddodLink.do?id=T14998987 |
인쇄
No. | 등록번호 | 청구기호 | 소장처 | 도서상태 | 반납예정일 | 예약 | 서비스 | 매체정보 |
---|---|---|---|---|---|---|---|---|
1 | WE00026156 | 576 | 가야대학교/전자책서버(컴퓨터서버)/ | 대출가능 |