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020 ▼a 9780438029019
035 ▼a (MiAaPQ)AAI10812251
035 ▼a (MiAaPQ)ku:15841
040 ▼a MiAaPQ ▼c MiAaPQ ▼d 248032
0820 ▼a 616.079
1001 ▼a Machacek, Miranda. ▼0 (orcid)0000-0003-1067-0561
24510 ▼a Elevated O-GlcNAcylation Enhances Pro-Inflammatory Th17 Function by Altering the Lipid Microenvironment.
260 ▼a [S.l.] : ▼b University of Kansas., ▼c 2018
260 1 ▼a Ann Arbor : ▼b ProQuest Dissertations & Theses, ▼c 2018
300 ▼a 80 p.
500 ▼a Source: Dissertation Abstracts International, Volume: 79-10(E), Section: B.
500 ▼a Advisers: Patrick Fields
5021 ▼a Thesis (Ph.D.)--University of Kansas, 2018.
520 ▼a Chronic inflammation is a feature of obesity and enhances the risk of atherosclerosis, cancer, diabetes, and autoimmunity. Specifically, pro-inflammatory Th17 and Th1 CD4+ effector T cells are increased in metabolic diseases. However, a clear mo
590 ▼a School code: 0099.
650 4 ▼a Immunology.
650 4 ▼a Cellular biology.
650 4 ▼a Biochemistry.
690 ▼a 0982
690 ▼a 0379
690 ▼a 0487
71020 ▼a University of Kansas. ▼b Pathology & Laboratory Medicine.
7730 ▼t Dissertation Abstracts International ▼g 79-10B(E).
773 ▼t Dissertation Abstract International
790 ▼a 0099
791 ▼a Ph.D.
792 ▼a 2018
793 ▼a English
85640 ▼u http://www.riss.kr/pdu/ddodLink.do?id=T14998017 ▼n KERIS
980 ▼a 201812 ▼f 2019
990 ▼a 관리자