| LDR | | 00000nmm u2200205 4500 |
| 001 | | 000000331622 |
| 005 | | 20241119163423 |
| 008 | | 181129s2018 ||| | | | eng d |
| 020 | |
▼a 9780438312784 |
| 035 | |
▼a (MiAaPQ)AAI10931514 |
| 035 | |
▼a (MiAaPQ)wustl:12610 |
| 040 | |
▼a MiAaPQ
▼c MiAaPQ
▼d 248032 |
| 049 | 1 |
▼f DP |
| 082 | 0 |
▼a 576 |
| 100 | 1 |
▼a Huynh, Jeremy Peter.
▼0 (orcid)0000-0002-1428-0686 |
| 245 | 10 |
▼a Identification of Bhlhe40 and Irg1 as Essential Regulators of the Inflammatory Response to Mycobacterium tuberculosis. |
| 260 | |
▼a [S.l.] :
▼b Washington University in St. Louis.,
▼c 2018 |
| 260 | 1 |
▼a Ann Arbor :
▼b ProQuest Dissertations & Theses,
▼c 2018 |
| 300 | |
▼a 155 p. |
| 500 | |
▼a Source: Dissertation Abstracts International, Volume: 80-01(E), Section: B. |
| 500 | |
▼a Adviser: Christina L. Stallings. |
| 502 | 1 |
▼a Thesis (Ph.D.)--Washington University in St. Louis, 2018. |
| 520 | |
▼a Protective immune responses to Mycobacterium tuberculosis (Mtb) must induce bactericidal functions while minimizing damage to the lung. Such responses require precise control of pro- and anti-inflammatory factors to regulate the recruitment and |
| 520 | |
▼a We utilized genetically deficient mice to assess the roles of Bhlhe40 and Irg1 expression on control of in vivo Mtb infection. We found that Bhlhe40 enables protective immune responses to Mtb by restricting expression of the anti-inflammatory cy |
| 590 | |
▼a School code: 0252. |
| 650 | 4 |
▼a Microbiology. |
| 650 | 4 |
▼a Immunology. |
| 690 | |
▼a 0410 |
| 690 | |
▼a 0982 |
| 710 | 20 |
▼a Washington University in St. Louis.
▼b Biology & Biomedical Sciences (Molecular Microbiology & Microbial Pathogenesis). |
| 773 | 0 |
▼t Dissertation Abstracts International
▼g 80-01B(E). |
| 773 | |
▼t Dissertation Abstract International |
| 790 | |
▼a 0252 |
| 791 | |
▼a Ph.D. |
| 792 | |
▼a 2018 |
| 793 | |
▼a English |
| 856 | 40 |
▼u http://www.riss.kr/pdu/ddodLink.do?id=T15001044
▼n KERIS |
| 980 | |
▼a 201812
▼f 2019 |
| 990 | |
▼a 관리자
▼b 관리자 |