| LDR | | 00000nmm u2200205 4500 |
| 001 | | 000000330411 |
| 005 | | 20241029165927 |
| 008 | | 181129s2018 ||| | | | eng d |
| 020 | |
▼a 9780438260689 |
| 035 | |
▼a (MiAaPQ)AAI10844660 |
| 035 | |
▼a (MiAaPQ)wisc:15564 |
| 040 | |
▼a MiAaPQ
▼c MiAaPQ
▼d 248032 |
| 049 | 1 |
▼f DP |
| 082 | 0 |
▼a 618 |
| 100 | 1 |
▼a Zou, Qingyun. |
| 245 | 14 |
▼a The Roles of GNA11 and GNA14 in Mediating FGF2- and VEGFA-Induced Endothelial Function Under Physiological Chronic Normoxia. |
| 260 | |
▼a [S.l.] :
▼b The University of Wisconsin - Madison.,
▼c 2018 |
| 260 | 1 |
▼a Ann Arbor :
▼b ProQuest Dissertations & Theses,
▼c 2018 |
| 300 | |
▼a 151 p. |
| 500 | |
▼a Source: Dissertation Abstracts International, Volume: 79-12(E), Section: B. |
| 500 | |
▼a Adviser: Jing Zheng. |
| 502 | 1 |
▼a Thesis (Ph.D.)--The University of Wisconsin - Madison, 2018. |
| 520 | |
▼a Background: During pregnancy, remarkable fetoplacental vascular growth and development occur to support the growing fetus. Interruption of such processes may cause adverse pregnancy outcomes such as intrauterine growth restriction and preeclamps |
| 590 | |
▼a School code: 0262. |
| 650 | 4 |
▼a Obstetrics. |
| 650 | 4 |
▼a Endocrinology. |
| 690 | |
▼a 0380 |
| 690 | |
▼a 0409 |
| 710 | 20 |
▼a The University of Wisconsin - Madison.
▼b Endocrinology and Reproductive Physiology. |
| 773 | 0 |
▼t Dissertation Abstracts International
▼g 79-12B(E). |
| 773 | |
▼t Dissertation Abstract International |
| 790 | |
▼a 0262 |
| 791 | |
▼a Ph.D. |
| 792 | |
▼a 2018 |
| 793 | |
▼a English |
| 856 | 40 |
▼u http://www.riss.kr/pdu/ddodLink.do?id=T15000003
▼n KERIS |
| 980 | |
▼a 201812
▼f 2019 |
| 990 | |
▼a 관리자
▼b 관리자 |